Bactrim hyperkalemia mechanism

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Actual: Medscape - Indication-specific dosing for Bactrim, Bactrim DS, Septra, Septra Treatment with recommended doses may cause hyperkalemia when trimethoprim is
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Hyperkalemia can be a side effect of the antibiotic Bactrim. monitoring antibiotics pharmacist Comments.

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6. Bactrim: the hyperkalemia induced by Bactrim is via an ENaC inhibitory effect exerted by the trimethoprim moiety. Pentamidine induced hyperkalemia via a similar mechanism. 7. calcineurin inhibitors (e.g, cyclosporine, tacrolimus): it is postulated that these medications inhibit renal tubular responsiveness to aldosterone. 8.

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The increase in creatinine associated with TMP is mild: 10% and reversible with drug discontinuation. The mechanism of Bactrim induced hyperkalemia is via Trimethoprim inhibition of the sodium channel located on the luminal surface of the principal cells, independent of aldosterone blockade.

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Hyperkalemia Hypernatremia Oocysts may also be seen on duodenal biopsy. Management: Bactrim; pyrimethamine leucovorin if Bactrim is contraindicated.

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The mechanism of Bactrim induced hyperkalemia is via Trimethoprim inhibition of the sodium channel located on the luminal surface of the principal cells, independent of aldosterone blockade. ( Same site of action of amiloride, triameterene and pentamidine).

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Hyperkalemia (Generally At High Dosages); Hypoglycemia (Rare); Hyponatremia Bactrim; Bactrim DS; Sulfatrim Pediatric; APO-Sulfatrim; Protrin DF; Septra

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6. Bactrim: the hyperkalemia induced by Bactrim is via an ENaC inhibitory effect exerted by the trimethoprim moiety. Pentamidine induced

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Hence, for Bactrim. - ↑ risk of hyperkalemia, especially with concomitant ACE-I, ARB and spironolactone. - Risk ↑ with higher doses of Bactrim. - Associated

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The increase in creatinine associated with TMP is mild: 10% and reversible with drug discontinuation. The mechanism of Bactrim induced hyperkalemia is via Trimethoprim inhibition of the sodium channel located on the luminal surface of the principal cells, independent of aldosterone blockade.

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